Lilacs for Alzheimer’s by meryddian.
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In Support of “Match it for Pratchett” () — helping raise money for Alzheimer’s research.
Quote from Terry Pratchett:
“I have had Alzheimer’s now for the past two years plus. There’s nearly as many of us as there are cancer sufferers, and it looks as if the number of people with the disease will double within a generation. It’s a shock and a shame to find out that funding for research is 3% of that which goes to find cancer cures.” – Terry Pratchett OBE, Patron, Alzheimer’s Research Trust
March 12, 2009 — A new product deemed a medical food by the Food and Drug Administration is now available in the United States, the manufacturer, Accera, has announced.
Known by the brand name Axona, the food targets metabolic deficiencies associated with Alzheimer’s disease (AD). It has been shown in a phase 2 clinical trial to improve cognition and memory in patients with mild to moderate disease, a statement from the company notes. Dispensed by prescription, it comes in single-serving packets and is taken once daily mixed with water.
Memories from people with Alzheimer’s assembled in quilt….
Nov. 29, 2009. A new study by a Swiss institutes finds a correlation between a diet rich in fat, sugar and cholesterol with the formation of the plaque identified in the brains of individuals diagnosed with dementias.
“On examining the brains of these mice, we found a chemical change not unlike that found in the Alzheimer brain,” Susanne Akterin, a researcher at the Karolinska Institutet’s Alzheimer’s Disease Research Center, who led the study, said in a statement.
Research in treatment is now focusing on the tangles caused by a buildup of the protein tau. In May’s Nature News Neurology, researchers describe how The amyloid-β peptide (Aβ) “interacts with the signalling pathways that regulate the phosphorylation of the microtubule-associated protein tau. Hyperphosphorylation of tau disrupts its normal function in regulating axonal transport and leads to the accumulation of neurofibrillary tangles and toxic species of soluble tau.”
Nov. 18, 2008. Small clusters of amyloid beta protein more influential in brain deterioratin than large plaque formations.
Scientists at the annual meeting of the Society for Neuroscience in Washington, D.C., say clusters of just two or three amyloid beta protein molecules can damage synapses, the connections between brain cells.
New research also shows that plaques of amyloid beta — much larger groupings made up of millions of cells — are not nearly as harmful as the small clusters.
A latest study led by Dr. Ville Leinonen of the University of Kuopio in Finland has found that an imaging method known as a PETdefine scan may help doctors in detecting “plaques” in the brain. These are an important factor in detecting Alzheimer’s disease.
Stem cells from an Alzheimer’s patient’s hip used to aide in regenerating neurons.
August 14, 2008 (Computerworld) University researchers in England are working to find out how the brain learns and stores memories by controlling a robot with a biological brain.
Scientists at the University of Reading today announced that they have developed a robot that is controlled by a biological brain formed by cultured neurons that were allowed to grow, divide and connect in a laboratory. Researchers are hoping that by watching how the brain learns, and how it stores and accesses memories, they will better understand diseases like Alzheimer’s and Parkinson’s, as well as strokes and brain injuries.